AQP

Since the discovery of first water channel ( AQP1) from red blood cell membrane by Agre et al in 1992, rapid and serial progresses have been made in characterization of AQP structure and function.

自从Agre等于1992年从红细胞膜发现第一个水通道蛋白AQP1以来，有关水通道蛋白结构与功能的研究取得了迅速的、系列性的进展。

The Expression of AQP-4 in Hippocampus of Rats after Moderate Traumatic Brain Injury Following Acute Alcoholism

急性酒精中毒合并中度创伤性脑损伤大鼠海马AQP4的表达

AQP 2 located in stria vascularis , Corti's organ , spiral ganglion and endolymphatic sac.

AQP2表达在血管纹 、 corti器 、 螺旋神经节细胞和内淋巴囊中.

Participates in assigned AQP meetings.

参与指定的AQP会议.

Conclusion AQP-1 may take part in the maintenance of the structural stability of the spiral ligament.

结论水通道蛋白-1可能参与维持耳蜗螺旋韧带处结构的稳定性。

The nano pump can convert the energy of the oscillating charge into the unidirectional transport of the water molecules. The maximal value of the water flux even exceeds the measured value for aquaporin-1 ( AQP-1).

该纳米泵可以把电荷振荡的能量转化为水分子的定向运动，流量最大时比细胞膜上的水通道蛋白（AQP-1）还要大。

Participate in the AQP meetings.

参加保证产品质量会议.

INTERVENTIONS: Cerebral ischemia model of rat was built according to the Longa's method. Haematoxylin and eosin staining was used to observe the histopathologic changes while immunohistochemical staining was used to detect the change of AQP 4 in brain tissue.

干预：参考Longa等方法建立大鼠大脑缺血模型，苏木精-伊红染色观察组织病理学改变，应用免疫组化染色检测脑组织中AQP-4的变化。

Aqp 3 in the spiral limb, vestibular lip, internal and external sulci, basilar membrane and basilar membrane crest, endolymphatic sac and duct, membranous semicircular canal and utricle and saccular macula;

Aqp3在螺旋韧带、前庭唇、内、外螺旋沟、基底膜和基底膜嵴、内淋巴囊和内淋巴管、膜半规管和椭圆囊、球囊斑等处显示荧光染色。

Methods AT ⅱ cells were isolated and purified from rats with ARDS, and the distribution of aquaporin-1 ( AQP-1) on the cells was observed by immunocytochemistry, immunoelectron microscopy and Western blotting.

方法急性分离纯化油酸ARDS模型大鼠的ATⅡ细胞，用免疫细胞化学、免疫电镜以及WESTERN BLOTTING方法测定AQP1的表达；

Conclusions Early hyperbaric oxygenation could decrease the expression levels of AQP-4 and caspase-3, and ameliorate cytotoxic brain edema and decrease the apoptosis of neurocytes, which may provide the neuroprotective mechanism against brain traumas.

结论早期高压氧治疗能减少大鼠脑外伤后AQP-4和caspase-3的表达，高压氧对创伤性脑损伤的脑保护机制可能与减轻神经胶质细胞介导的细胞性脑水肿、减少神经细胞凋亡有关。

Leads internal AQP teams in launching of new products.

在投入新产品时领导内部团队.